Tehami Dermatology
    Pediatric dermatology

    Eczema in children: a practical plan for parents

    Steroid creams are safe when used correctly, and moisturiser quantity matters more than the brand. A clear routine for families.

    6 min readUpdated: 2026-09-01By Dr. Mohammed Tehami
    Mother applying moisturiser to a young child

    Atopic eczema affects a large share of children, and most families arrive exhausted from broken sleep and scratching. It is a condition of a leaky skin barrier and an over-reactive immune response. It is not an allergy to be found and removed, and it is not something a child simply has to endure.

    Key points

    • Moisturise generously two to three times a day, every day
    • Steroid creams are safe used properly and stopped when clear
    • Fingertip units make dosing simple and accurate
    • Do not remove foods without proper allergy assessment
    • Sudden worsening with crusting suggests infection

    The daily routine that does most of the work

    Bathe once daily in lukewarm water for five to ten minutes with a soap-free cleanser, then pat dry and apply moisturiser within three minutes. Quantity is what matters: a young child needs roughly 250 grams of emollient per week, applied two or three times daily even when the skin looks calm.

    Choose a thick ointment or cream rather than a lotion. Avoid perfumed products, bubble baths and antiseptic additives, all of which irritate.

    Using steroid creams correctly

    Fear of steroid creams is the main reason eczema stays uncontrolled. Used properly they are safe and they end flares quickly. Apply the strength your doctor prescribed to inflamed skin once or twice daily until the skin is smooth, usually seven to fourteen days, then stop the steroid and continue moisturiser.

    Measure with the fingertip unit: the amount squeezed from the fingertip to the first crease treats an area the size of two adult palms. For children with repeated flares in the same places, applying treatment twice weekly to those areas prevents relapse. Non-steroid options such as calcineurin inhibitors are useful on the face and eyelids.

    Triggers worth controlling

    Heat and sweating, dust, wool fabrics, harsh detergents and dry indoor air from air conditioning all provoke flares. Keep nails short, use cotton clothing, rinse laundry well, and cool the room at night.

    Food allergy is a genuine cause in a minority of infants with severe eczema, usually alongside immediate reactions such as hives or vomiting. Removing foods without evidence risks nutritional harm and rarely improves the skin.

    When to seek help quickly

    Weeping, yellow crusting, painful clustered blisters, fever, or eczema that no longer responds to a treatment that used to work all suggest infection and need prompt review. Severe eczema that disrupts sleep and school deserves specialist assessment, since effective systemic and biologic treatments now exist.

    Frequently asked questions

    Will my child grow out of eczema?

    Many children improve markedly by school age, and a proportion continue with sensitive or dry skin into adulthood. Good control in childhood reduces flares and scarring from scratching.

    Can eczema be managed by video consultation?

    Yes. Photographs plus a detailed history allow diagnosis and a full written routine, and follow up by video works well for adjusting treatment.

    Step by step treatment pathway

    Atopic eczema

    A control pathway built on barrier repair, anti-inflammatory treatment matched to severity, and prompt recognition of infection.

    Step 1: assessment and diagnosis

    • Grade severity with a validated score such as SCORAD or EASI, and record sleep loss and itch, which matter more to families than surface area.
    • Map the distribution: face and scalp in infants, flexures in older children, hands and eyelids in adults.
    • Look for infection: weeping, golden crust, pustules, or a sudden flare that will not settle [1].
    • Ask about triggers: heat and sweating, dust, soap, wool, and in a minority, specific foods.

    Step 2: grading severity

    Mild

    Dry skin with limited areas of itch and redness, minimal sleep disturbance.

    Emollients plus a mild topical steroid for flares.

    Moderate

    Frequent flares, excoriation, disturbed sleep, thickened skin.

    Potent topical steroid short courses, calcineurin inhibitor for the face, proactive weekend therapy.

    Severe

    Widespread inflammation, constant itch, infection, major impact on school, work or sleep.

    Specialist systemic therapy assessment.

    Step 3: treatment in sequence

    First line

    • Generous emollient use, applied several times daily and continued when the skin is clear [4].
    • Soap substitutes, short lukewarm showers, cotton clothing, nails cut short.
    • Topical corticosteroid matched to site and severity: mild on the face and folds, moderate to potent on the body, used in short bursts until clear [1,2].
    • Treat infection when present rather than escalating steroid strength.

    Second line

    • Topical calcineurin inhibitor (tacrolimus, pimecrolimus) for the face, eyelids and folds, and as a steroid sparing maintenance agent.
    • Proactive therapy: apply treatment twice weekly to sites that always flare, which reduces relapse rates [1].
    • Narrowband UVB phototherapy for widespread disease not controlled by topicals.
    • Address the bacterial load with antiseptic measures and a targeted antibiotic only when clinically infected. Repeated antibiotic courses are avoided.

    Third line and refractory disease

    • Systemic therapy under specialist care: ciclosporin for rapid control, methotrexate or azathioprine for maintenance.
    • Biologic therapy such as dupilumab, or a JAK inhibitor, where available and appropriate, with the relevant screening and monitoring.
    • Patch testing when eczema becomes resistant or changes distribution, because contact allergy is a common hidden driver.
    • Reassess the diagnosis: scabies, cutaneous T cell lymphoma and psoriasis all mimic resistant eczema.

    Special groups

    Infants and children

    Face and scalp involvement is normal in infancy. Food allergy testing is only indicated with a clear, reproducible reaction history, not for eczema alone [2]. Steroid phobia is addressed openly with written quantities.

    Pregnancy

    Emollients and topical steroids remain first line. Ciclosporin is the usual systemic choice when one is needed; methotrexate is contraindicated.

    Darker skin

    Redness may look violet or grey, eczema is often follicular, and both post-inflammatory hyperpigmentation and hypopigmentation are common and need explaining as temporary.

    Hot climate

    Sweat and dust are major triggers. Rinse after sweating, use light cotton layers, and use lighter lotions by day with richer ointments at night.

    When to investigate

    • Skin swab when infection is suspected or treatment is failing.
    • Viral swab for suspected eczema herpeticum, which is an emergency.
    • Patch testing for resistant or adult onset hand and face eczema.
    • Biopsy where lymphoma or another diagnosis is possible.

    When to refer

    • Eczema herpeticum, painful punched out erosions with fever: treat urgently and refer the same day.
    • Failure of correct topical treatment, or a need for systemic therapy.
    • Severe sleep loss, growth impact in a child, or recurrent infection.

    Review interval

    Review at 2 to 4 weeks during a flare, then 3 to 6 monthly for maintenance and treatment quantity checks.

    Pathway sources

    1. [1] American Academy of Dermatology, atopic dermatitis guidelines
    2. [2] NICE guideline CG57, atopic eczema in under 12s
    3. [3] DermNet, atopic dermatitis
    4. [4] Cochrane review, emollients and moisturisers for eczema

    Updated: 2026-09-12

    This pathway summarises recommended practice for education. It is not a personal prescription. Actual treatment is decided by a doctor after examination.

    Need advice for your own skin?

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